Samuel G Horst

This study identifies fibrillin 3 as a target of Shh/Smo signaling in zebrafish fin regeneration, showing its role in ray branching through microfibril dynamics and cell interactions.

The paper shows early exercise during zebrafish fin regeneration disrupts an extracellular matrix program essential for tissue repair.

This study uses zebrafish fras1 mutants to investigate Fraser Syndrome's role in fin skeletal patterning, revealing defects in basal epidermis and osteoblast integration during development and regeneration.

The paper uses single-cell RNA sequencing to analyze zebrafish ovarian cells, identifying novel cell subpopulations and gene functions involved in ovarian development.