Author

Yuejin Wu

Johns Hopkins UniversityORCID 0000-0003-3547-8082Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Elizabeth D. LuczakiD· 2 shared Jonathan M. GrangeriD· 2 shared Kevin R. MurphyiD· 2 shared Mark E. AndersoniD· 2 shared Priya UmapathiiD· 2 shared Adam G. Rokita· 1 shared Albert J. R. HeckiD· 1 shared Amin SabetiD· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersMolecular Biology

Elizabeth D. LuczakORCID

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2 papers

Recent·Most cited

Molecular BiologyNature CommunicationsPeer-reviewedSep 4, 2020

Mitochondrial CaMKII causes metabolic reprogramming, energetic insufficiency, and dilated cardiomyopathy

Elizabeth D. Luczak, Yuejin Wu, Jonathan M. Granger…·Mark E. Anderson

This study shows mitochondrial CaMKII activation leads to metabolic reprogramming, energetic insufficiency, and dilated cardiomyopathy, with potential therapeutic targets identified.

94 citationstop 4%

mouseexperimental assay

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Molecular BiologybioRxivPreprintFeb 18, 2020

O-GlcNAcylation, oxidation and CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes by distinct mechanisms

Olurotimi O. Mesubi, Adam G. Rokita, Neha Abrol…·Mark E. Anderson

This study shows that O-GlcNAcylation and oxidative stress via CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes through distinct mechanisms.

1 citation

mouseexperimental assay

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