Author

Priya Umapathi

Johns Hopkins UniversityORCID 0000-0002-1872-6753Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3

Frequent Co-Authors

Elizabeth D. LuczakiD· 3 shared Jonathan M. GrangeriD· 3 shared Mark E. AndersoniD· 3 shared An‐Chi WeiiD· 2 shared Gerald Warren HartiD· 2 shared Kevin R. MurphyiD· 2 shared Natasha Elizabeth ZacharaiD· 2 shared Neha Abrol· 2 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersMolecular Biology

Elizabeth D. LuczakORCID

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3 papers

Recent·Most cited

Excessive O-GlcNAcylation causes heart failure and sudden death

Priya Umapathi, Partha Sarathi Banerjee, Natasha Elizabeth Zachara, Neha Abrol, Qinchuan Wang, Olurotimi O. Mesubi, Elizabeth D. Luczak, Yuejin Wu, Jonathan M. Granger, An‐Chi Wei, Oscar E. Reyes Gaido, Liliana Florea, C. Conover Talbot, Gerald Warren Hart, Mark E. Anderson

This paper shows that excessive O-GlcNAcylation causes heart failure and sudden death in mice, while reducing it protects against cardiac stress.

Circulation · Feb 17, 2021Peer-reviewedMolecular Biology

Mitochondrial CaMKII causes metabolic reprogramming, energetic insufficiency, and dilated cardiomyopathy

Elizabeth D. Luczak, Yuejin Wu, Jonathan M. Granger, Mei-ling A. Joiner, Nicholas R. Wilson, Ashish K Gupta, Priya Umapathi, Kevin R. Murphy, Oscar E. Reyes Gaido, Amin Sabet, Eleonora Corradini, Wen-Wei Tseng, Yibin Wang, Albert J. R. Heck, An‐Chi Wei, , Mark E. Anderson

This study shows mitochondrial CaMKII activation leads to metabolic reprogramming, energetic insufficiency, and dilated cardiomyopathy, with potential therapeutic targets identified.

Nature Communications · Sep 4, 2020Peer-reviewedMolecular Biology

O-GlcNAcylation, oxidation and CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes by distinct mechanisms

Olurotimi O. Mesubi, Adam G. Rokita, Neha Abrol, Yuejin Wu, Biyi Chen, Qinchuan Wang, Jonathan M. Granger, Anthony Tucker-Bartley, Elizabeth D. Luczak, Kevin R. Murphy, Priya Umapathi, Partha S. Banerjee, Tatiana N. Boronina, Robert Norman Cole, Lars Siegfried Maier, , Mark E. Anderson

This study shows that O-GlcNAcylation and oxidative stress via CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes through distinct mechanisms.

bioRxiv · Feb 18, 2020 · PreprintMolecular Biology