Author

J. David Sweatt

Vanderbilt UniversityORCID 0000-0003-3567-485XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Andrew John KennedyiD· 2 shared Allie JiniD· 1 shared Andrew E. JaffeiD· 1 shared BaDoi N. PhaniD· 1 shared Benjamin D. PhilpotiD· 1 shared Brady J. MaheriD· 1 shared Celeste B. GreeriD· 1 shared Courtney ThaxtoniD· 1 shared

Activity by Year

2017-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Andrew John KennedyORCID

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2 papers

Recent·Most cited

NeuroscienceJournal of NeurosciencePeer-reviewedDec 1, 2020

Tet1 isoforms differentially regulate gene expression, synaptic transmission and memory in the mammalian brain.

Celeste B. Greer, J. Wright, J.D. Weiss…·Garrett A. Kaas

This study identifies two Tet1 isoforms with distinct roles in gene expression, synaptic transmission, and memory in the mammalian brain.

41 citations

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintMay 11, 2017

Defects of myelination are common pathophysiology in syndromic and idiopathic autism spectrum disorders

BaDoi N. Phan, Stephanie C. Page, Morganne N. Campbell…·Brady J. Maher

The paper compares mouse models of syndromic ASD with human ASD brain data to identify shared myelination and axon development disruptions across both syndromic and idiopathic ASD.

3 citations

𝕏 PostBluesky