Takashi Kiuchi

This study identifies cis-regulatory changes in Wnt1 and Wnt6 as contributors to the caudal horn size difference between Bombyx mori and B. mandarina.

This study identifies the BmMasc-BmPSI complex as essential for male-type Bmdsx splicing in silkworms by binding to female-specific introns and exons.

This study shows that knocking out cryptochrome 1 in silkworms disrupts circadian rhythms and photoperiodic diapause induction, highlighting its role as a photoreceptor in the circadian clock.

This paper identifies Bmmamo as the gene responsible for the dilute black (bd) phenotype in silkworms, linking it to female sterility and larval pigmentation.

The paper identifies two recessive lethal mutations in semiconsomic silkworm strains, showing that deleterious mutations on a B. mandarina-derived chromosome cause embryonic lethality.

This study shows that knocking out core circadian clock genes in silkworms disrupts photoperiodic diapause induction during both embryonic and larval stages.

The paper investigates dosage compensation in silkworm males by analyzing allele-specific expression and finds both Z chromosomes are transcriptionally downregulated.

The paper analyzes transcriptome changes in silkworm larvae overexpressing a piRNA-resistant Masculinizer gene, finding global repression of Z-linked genes in females but minimal effect on males.

The paper uses CRISPR/Cas9 to knockout pigment-related genes in Bombyx mori and its wild ancestor, showing that apt-like contributes to reduced pigmentation during domestication.

The paper identifies a mutation in the Bmmah gene, a β-group solute carrier gene, as responsible for the reddish-brown egg and eye coloration in the silkworm b-4 mutant.

This study identifies CENP-T as essential for CenH3-independent kinetochore assembly in Lepidoptera, showing its role in recruiting the Mis12 complex and supporting mitotic progression.