Author

Tony L. Goldberg

University of Wisconsin–MadisonORCID 0000-0003-3962-4913Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genomics· 3 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 4 Organism:non-human primate· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 1 Assay:metagenomics· 1 Research Focus:phylogenetics / evolution· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Colin A. ChapmaniD· 2 shared David Hyeroba· 2 shared Geoffrey Weny· 2 shared Nelson TingiD· 2 shared Noah D. SimonsiD· 2 shared Adam James ReidiD· 1 shared Adam L. Bailey· 1 shared Ajay K. SethiiD· 1 shared

Activity by Year

2014-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersGenomics

Colin A. ChapmanORCID

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4 papers

Recent·Most cited

Genomicsnpj Antimicrobials and ResistancePeer-reviewedOct 29, 2026

Antimicrobial use contributes to resistance gene enrichment across cattle groups on commercial dairy farms

Andrew J. Steinberger, Colette A. Nickodem, Juliana Leite de Campos…·Jessica L. Hite

The paper links antimicrobial use (AMU) on dairy farms to variations in resistance gene profiles across cattle groups, showing that higher AMU correlates with increased resistance to certain drug classes.

DOI 10.1038/s44259-026-00291-8

metagenomicsstatistical model

𝕏 PostBluesky

GenomicsPLoS PathogensPeer-reviewedAug 3, 2020

Genomic and transcriptomic evidence for descent from Plasmodium and loss of blood schizogony in Hepatocystis parasites from naturally infected red colobus monkeys

Eerik Aunin, Ulrike Böhme, Theo Sanderson…·Adam James Reid

The paper presents genomic and transcriptomic evidence showing Hepatocystis parasites are closely related to Plasmodium but lack blood replication and have lost genes involved in red blood cell invasion.

34 citations

non-human primatewhole-genome sequencing

𝕏 PostBluesky

GenomicsGenome Biology and EvolutionPeer-reviewedMay 9, 2019

Genome-wide patterns of gene expression in a wild primate indicate species-specific mechanisms associated with tolerance to natural simian immunodeficiency virus infection

Noah D. Simons, Geeta N. Eick, María José Ruiz‐López…·Nelson Ting

The paper analyzes gene expression in wild Ugandan red colobus to show SIV infection does not cause chronic immune activation and identifies upregulation of CD101 as a potential tolerance mechanism.

10 citations

non-human primatebulk RNA-seq

𝕏 PostBluesky

MicrobiologyPLoS ONEPeer-reviewedMar 20, 2014

High Genetic Diversity and Adaptive Potential of Two Simian Hemorrhagic Fever Viruses in a Wild Primate Population

Adam L. Bailey, Michael Lauck, Andrea Weiler…·David H. O'Connor

The paper identifies two novel SHFVs in wild primates, showing high genetic diversity and signs of positive selection, suggesting adaptive potential.

DOI 10.1371/journal.pone.0090714

non-human primatestatistical model

𝕏 PostBluesky