Kazue Hashimoto‐Torii

The paper shows that ciliopathy and neonatal ketamine exposure interact to cause motor deficits through non-apoptotic caspase activation, which can be reversed by caspase inhibition.

This study shows that human cortical organoids generated across multiple sites using a harmonized protocol consistently produce similar cell types and structures, though with some site-specific differences in size and stress markers.

The paper investigates molecular diversity and connectivity patterns in accessory olfactory system neurons, revealing lineage- and sex-specific differences in connectivity.

This study identifies changes in fatty acid metabolism and gut microbiota in a PAE mouse model, linking them to neurobehavioral deficits and lipid droplet accumulation in the brain.

This study shows that placental allopregnanolone (ALLO) is critical for normal cerebellar development and social behavior in male mice, with ALLO deficiency leading to autism-like traits that can be rescued by ALLO treatment.