Author

Dorota Studniarczyk

University College LondonORCID 0000-0003-4109-7956Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:electronic health records· 1 Organism:mouse· 1

Frequent Co-Authors

Mark FarrantiD· 2 shared Ashton J. CurtisiD· 1 shared Cécile BatsiD· 1 shared Craig A. SextoniD· 1 shared Ian D. CoombsiD· 1 shared Jian Zhu· 1 shared Matthew G. GoldiD· 1 shared Stuart Graham Cull-CandyiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 20 mutual co-author links

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2 shared papersNeuroscience

Mark FarrantORCID

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2 papers

Recent·Most cited

NeuroscienceJournal of NeurosciencePeer-reviewedOct 23, 2025

Widely used CaMKII regulatory segment mutations cause tight actinin binding and dendritic spine enlargement in unstimulated neurons

Ashton J. Curtis, Jian Zhu, Dorota Studniarczyk…·Matthew G. Gold

This study shows that CaMKII T305A/T306A mutations cause tight actinin binding and dendritic spine enlargement in unstimulated neurons, suggesting a structural role for CaMKII in LTP.

DOI 10.1523/jneurosci.0795-25.2025

experimental assay

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NeuroscienceeLifePeer-reviewedApr 12, 2023

Intracellular NASPM allows an unambiguous functional measure of GluA2-lacking calcium-permeable AMPA receptor prevalence

Ian D. Coombs, Cécile Bats, Craig A. Sexton…·Mark Farrant

The paper introduces NASPM as a tool to unambiguously measure calcium-permeable AMPA receptor prevalence by blocking GluA1 currents regardless of auxiliary subunits.

26 citations

mouseelectronic health records

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