Author

Johanna Krüger

Oulu University HospitalORCID 0000-0003-4346-1133Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Johanna Krüger1 paper · Hertie Institute for Clinical Brain Research

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:benchmarking· 1 Research Focus:database / resource· 1 Assay:imaging· 1 Research Focus:software / tool· 1 Research Focus:spatial domain detection· 1 Approach:statistical model· 1

Frequent Co-Authors

Aleksi RasilaiD· 1 shared Anastasia ShakirzyanovaiD· 1 shared Annakaisa HaapasaloiD· 1 shared Anne M. PortaankorvaiD· 1 shared Anssi Koivula· 1 shared Ashutosh DhingraiD· 1 shared Dorit B. HoffmanniD· 1 shared Eila Korhonen· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Aleksi RasilaORCID

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2 papers

Recent·Most cited

NeuroscienceNeuroImagePeer-reviewedOct 1, 2025

Reduced harmonic complexity of brain parenchymal cardiovascular pulse waveforms in Alzheimer's disease

Anssi Koivula, Vilma Perkiömäki, Mika Aho…·Janne Kananen

This study introduces MREGRHP, a frequency domain imaging method, and finds reduced harmonic complexity in brain pulse waveforms in Alzheimer's disease patients.

3 citations

𝕏 PostBluesky

NeuroscienceMolecular PsychiatryPeer-reviewedSep 26, 2025

Frontotemporal dementia patient-derived iPSC neurons show cell pathological hallmarks and evidence for synaptic dysfunction and DNA damage

Nadine Huber, Tomi Hietanen, Sami Heikkinen…·Annakaisa Haapasalo

This study shows that iPSC-derived neurons from FTD patients exhibit synaptic dysfunction, DNA damage, and C9-HRE-related pathologies, supporting synaptic dysfunction as a key factor in FTD.

4 citations

humanexperimental assay

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