Author

Emmanuel Quansah

Van Andel InstituteORCID 0000-0003-4532-1724Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Jennifer A. SteineriD· 2 shared Martha L. Escobar GalvisiD· 2 shared Patrik BrundiniD· 2 shared Allison LindquistiD· 1 shared Carla Gilliland· 1 shared Daniel Saiz‐SáncheziD· 1 shared Diptaman ChatterjeeiD· 1 shared Elizabeth EnsinkiD· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Jennifer A. SteinerORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintAug 2, 2024

Tet2 loss suppress α-synuclein pathology by stimulating ciliogenesis

Emmanuel Quansah, Naman Vatsa, Elizabeth Ensink…·Juozas Gordevičius

The paper identifies that Tet2 loss promotes ciliogenesis and reduces α-synuclein pathology in Parkinson's disease models.

1 citation

mousebulk RNA-seq

𝕏 PostBluesky

NeuroscienceJournal of Parkinson s DiseasePeer-reviewedMar 29, 2019

Loss of one Engrailed1 allele enhances induced α-synucleinopathy

Diptaman Chatterjee, Daniel Saiz‐Sánchez, Emmanuel Quansah…·Patrik Brundin

This study shows that loss of one Engrailed1 allele in mice increases α-synuclein aggregation after PFF injection, suggesting a role for En1 in PD pathology.

16 citations

mouseexperimental assay

𝕏 PostBluesky