Matthew R. Jones

This study identifies Oncostatin M (OSM) as a critical regulator of pulmonary host responses during influenza and bacterial pneumonia, revealing non-canonical signaling pathways.

The paper defines sub-phenotypes of pneumonia using histopathological features from human lung samples and validates them in mouse models.

This study identifies lung CD4+ resident memory T cells and their interaction with airway secretory cells as key regulators of neutrophilic allergic airways disease in mice and humans.