Author

Kunihiro Matsumoto

Nagoya UniversityORCID 0000-0003-4821-0106Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:C. elegans· 1 Research Focus:cell-cell communication· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Naoki HisamotoiD· 2 shared Anindya Ghosh‐RoyiD· 1 shared Atrayee BasuiD· 1 shared Hiromu Shojima· 1 shared Hiroshi HanafusaiD· 1 shared Kiyohito MizutaniiD· 1 shared Kota Ohashi· 1 shared Muneaki Miyata· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPathology

Naoki HisamotoORCID

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2 papers

Recent·Most cited

PathologybioRxivPreprintOct 3, 2025

Astrocytic Cathepsin D truncates α-synuclein and promotes Lewy neurite-like aggregation in neurons

Hiroshi Hanafusa, Kota Ohashi, Hiromu Shojima…·Kunihiro Matsumoto

This study shows astrocytes process and propagate α-synuclein aggregates via lysosomal protease Cathepsin D, promoting Lewy neurite-like aggregation in neurons.

DOI 10.1101/2025.10.03.680233

humanproteomics

𝕏 PostBluesky

NeurosciencebioRxivPreprintNov 18, 2019

UNC-16/JIP3 negatively regulates actin dynamics dependent on DLK-1 and microtubule dynamics independent of DLK-1 in regenerating neurons

Sucheta S. Kulkarni, Vidur Sabharwal, Seema Sheoran…·Sandhya Padmanabhan Koushika

This study shows UNC-16/JIP3 inhibits axonal regeneration by regulating DLK-1 function, affecting actin and microtubule dynamics differently depending on DLK-1 involvement.

2 citations

C. elegansexperimental assay

𝕏 PostBluesky