Author

Álvaro Viedma-Poyatos

Consejo Superior de Investigaciones CientíficasORCID 0000-0003-4920-6328Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

Dolores Pérez‐SalaiD· 1 shared Elena Hernández-Gerez· 1 shared María A. Pajares· 1 shared Nuria Goya‐IglesiasiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

4 top collaborators · 6 mutual co-author links

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1 shared paperCell Biology

Dolores Pérez‐SalaORCID

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1 paper

Recent·Most cited

Cell BiologyFree Radical Biology and MedicinePeer-reviewedMay 6, 2026

Lysosomal function, resistance to stress and repair are compromised by expression of the Alexander disease GFAP R239C mutant

Elena Hernández-Gerez, Nuria Goya‐Iglesias, Álvaro Viedma-Poyatos…·Dolores Pérez‐Sala

This study shows that expressing the GFAP R239C mutant in astrocytes disrupts lysosomal function, acidification, and repair, contributing to proteostasis defects and neurodegeneration in Alexander disease.

1 citation

humanproteomics

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