Author

Artha A. Lam

University of California, RiversideORCID 0009-0001-1023-5204Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Anthony E. BishayiD· 2 shared Crystal N. LunaiD· 2 shared Elena V. KozlovaiD· 2 shared Margarita Currás‐CollazoiD· 2 shared Maximillian E. DenysiD· 2 shared Amna Habbal· 1 shared Anastasia Ghilenschi ColtoniD· 1 shared Ansel HsiaoiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersNeuroscience

Anthony E. BishayORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintJul 22, 2026

Social and neuroendocrine phenotypes reprogrammed by endocrine-disrupting chemicals can be mitigated by Limosilactobacillus reuteri modulation of the gut microbiome-thyroid-oxytocin axis

Elena V. Kozlova, Maximillian E. Denys, Anthony E. Bishay…·Margarita Currás‐Collazo

This study shows that Limosilactobacillus reuteri supplementation mitigates PBDE-induced ASD-like social and neuroendocrine deficits in mice by modulating the gut microbiome-thyroid-oxytocin axis.

DOI 10.64898/2026.07.18.739173

mousebulk RNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintJul 28, 2025

Maternal Thyroid Supplementation Prevents Autistic-relevant Social Behavior and Hypothalamic Oxytocin Depletion Produced by Developmental Exposure to Environmental Toxicants

Elena V. Kozlova, Maximillian E. Denys, Anthony E. Bishay…·Margarita Currás‐Collazo

This study shows maternal thyroid supplementation prevents PBDE-induced social behavior deficits and hypothalamic oxytocin depletion in mice.

3 citations

mousestatistical model

𝕏 PostBluesky