Anthony E. Bishay

This study shows that early-life exposure to PBDEs in mice leads to autism-like behaviors and changes in prosocial neuropeptide expression.

This study shows that Limosilactobacillus reuteri supplementation mitigates PBDE-induced ASD-like social and neuroendocrine deficits in mice by modulating the gut microbiome-thyroid-oxytocin axis.

This study shows maternal thyroid supplementation prevents PBDE-induced social behavior deficits and hypothalamic oxytocin depletion in mice.

This study shows that exposure to PBDE mixture DE-71 during pregnancy and lactation in female mice leads to autism-relevant behavioral and neurochemical changes in offspring.