Margarita Currás‐Collazo

Enduring Autism-like Phenotypes and Deregulated Hypothalamic Prosocial Peptides After Early-Life Exposure to Indoor Flame Retardants in Male C57BL/6 Mice

This study shows that early-life exposure to PBDEs in mice leads to autism-like behaviors and changes in prosocial neuropeptide expression.

Social and neuroendocrine phenotypes reprogrammed by endocrine-disrupting chemicals can be mitigated by Limosilactobacillus reuteri modulation of the gut microbiome-thyroid-oxytocin axis

This study shows that Limosilactobacillus reuteri supplementation mitigates PBDE-induced ASD-like social and neuroendocrine deficits in mice by modulating the gut microbiome-thyroid-oxytocin axis.

Maternal Thyroid Supplementation Prevents Autistic-relevant Social Behavior and Hypothalamic Oxytocin Depletion Produced by Developmental Exposure to Environmental Toxicants

This study shows maternal thyroid supplementation prevents PBDE-induced social behavior deficits and hypothalamic oxytocin depletion in mice.

Persistent autism-relevant phenotype produced by in utero and lactational exposure of female mice to the commercial PBDE mixture, DE-71

This study shows that exposure to PBDE mixture DE-71 during pregnancy and lactation in female mice leads to autism-relevant behavioral and neurochemical changes in offspring.