Author

Zhihong Lin

Duke UniversityORCID 0009-0002-0144-8336Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:proteomics· 1

Frequent Co-Authors

Beverly deSouza· 1 shared Eric Adams· 1 shared Gloria S. LeeiD· 1 shared Guanghao LiuiD· 1 shared Jacob RystediD· 1 shared John A. WemmieiD· 1 shared Kristin A. Anderson· 1 shared Matthew D. HirscheyiD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMolecular Biology

Beverly deSouza

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2 papers

Recent·Most cited

SIRT4 Controls Macrophage Function and Wound Healing through Control of Protein Itaconylation in Mice

Kristin A. Anderson, Beverly deSouza, Pol Castellano‐Escuder, Zhihong Lin, Olga Ilkayeva, Michael J. Muehlbauer, Paul A. Grimsrud, Matthew D. Hirschey

This study identifies SIRT4 as a lysine deitaconylase that regulates macrophage inflammatory responses and wound healing by modulating protein itaconylation in mice.

bioRxiv · May 12, 2025 · PreprintMolecular Biology

Loss of tau and Fyn reduces compensatory effects of MAP2 for tau and reveals a Fyn-independent effect of tau on glutamate-induced Ca2+ response

Guanghao Liu, Ramasamy Thangavel, Jacob Rysted, Yohan Kim, Meghan B. Francis, Eric Adams, Zhihong Lin, Rebecca J. Taugher, John A. Wemmie, Yuriy M. Usachev, Gloria S. Lee

The study investigates the role of tau and Fyn interaction in glutamate-induced Ca2+ response using a tau/Fyn double knockout mouse, revealing a Fyn-independent effect of tau and MAP2's compensatory role.

Journal of Neuroscience Research · Aug 26, 2019Peer-reviewedNeuroscience