Author

Sarahi G. Molina

University of North Carolina at Chapel HillORCID 0009-0002-1602-3453Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Adriana S. BeltrániD· 1 shared Attila SzénásiiD· 1 shared Austin A. WhitmaniD· 1 shared Charles M. PerouiD· 1 shared Christine ThaiiD· 1 shared Denis O. OkumuiD· 1 shared Elizabeth BrunkiD· 1 shared Gary L. JohnsoniD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Adriana S. BeltránORCID

Mutual co-authors in cluster

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1 paper

Recent·Most cited

Kinase Plasticity in Response to Vandetanib Enhances Sensitivity to Tamoxifen in Estrogen Receptor Positive Breast Cancer

Austin A. Whitman, Rasha T. Kakati, Santiago Haase, Christine Thai, Surya Kant Tripathi, Attila Szénási, Elizabeth Brunk, Denis O. Okumu, Michael P. East, Sarahi G. Molina, Adriana S. Beltrán, Jesse R. Raab, LA Carey, Charles M. Perou, Gary L. Johnson, Philip M. Spanheimer

This study shows vandetanib enhances tamoxifen sensitivity in ER+ breast cancer by reprogramming ER gene regulation and kinase networks, identifying adaptive resistance pathways and response signatures.

Molecular Cancer Therapeutics · Jul 28, 2026Peer-reviewedCancer Biology