Author

Andrea Laganson

The Ohio State University Comprehensive Cancer Center – Arthur G. James Cancer Hospital and Richard J. Solove Research InstituteORCID 0009-0002-9128-7598Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Ann‐Kathrin EisfeldiD· 2 shared Jill L. Buss· 2 shared Sanam LoghaviiD· 2 shared Adrienne Heyrosa· 1 shared Alan H. ShihiD· 1 shared Alice S. MimsiD· 1 shared Andrew J. CarrolliD· 1 shared Andrew R. StiffiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

Ann‐Kathrin EisfeldORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Prognostic and Therapeutic Implications of BRAF Mutations in Acute Myeloid Leukemia

Darren Lee, Yazan Abu-Shihab, Kate Plas, Deedra Nicolet, Krzysztof Mrózek, Mark J. Routbort, Keyur Pravinchandra Patel, Christopher James Walker, Jill L. Buss, Andrew R. Stiff, Andrea Laganson, Courtney Denton Dinardo, Naval Guastad Daver, Tapan Mahendra Kadia, Farhad Ravandi, , Linde A. Miles

This study identifies BRAF mutations in AML, showing poor outcomes and unique co-mutation patterns, suggesting potential targeted therapies.

Leukemia · Jun 15, 2026Peer-reviewedCancer Biology

Multiomic characterization, early detection, and therapeutic targeting of myeloid sarcoma

Bettina Nadorp, Audrey Lasry, Sanam Loghavi, Ravi Patel, Hager M. Mansour, Benjamin Kelly, Christopher J. Walker, Jill L. Buss, Isaiah Boateng, Wafa Al-Santli, Zoe B. Ciantra, Rebecca J. Austin, Adrienne Heyrosa, Helee Desai, Andrea Laganson, , Ann‐Kathrin Eisfeld

This study characterizes myeloid sarcoma through multi-omic profiling, identifies RAS pathway activation as a hallmark, and shows RAS inhibition reduces tumor burden in mouse models.

bioRxiv · Dec 5, 2025 · PreprintCancer Biology