Author

Christopher J. DiRusso

Boston UniversityORCID 0009-0003-7143-2572Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Research Focus:clustering· 1 Organism:mouse· 1 Assay:multiome· 1

Frequent Co-Authors

Thomas D. GilmoreiD· 3 shared Wei WangiD· 2 shared Adrian WhittyiD· 1 shared Aidan T. Lackstrom· 1 shared Alan T. YeoiD· 1 shared Alejandra Navarro-Rosado· 1 shared Alexandra V. Grigoryeva· 1 shared Angela Calderon· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersMolecular Biology

Thomas D. GilmoreORCID

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3 papers

Recent·Most cited

Molecular BiologyComparative Immunology ReportsPeer-reviewedJun 17, 2025

Comparison of Activities of Transcription Factor NF-κB from Two Jellyfish Models

Leah M. Williams, Wei Wang, Alexandra V. Grigoryeva…·Thomas D. Gilmore

This study compares NF-κB activity in two jellyfish species, showing differences in transcriptional activation and subcellular localization.

1 citation

proteomicsmultiome

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Molecular BiologyJournal of Biological ChemistryPeer-reviewedOct 27, 2023

A Conserved Core Region of the Scaffold NEMO is Essential for Signal-induced Conformational Change and Liquid-liquid Phase Separation

Christopher J. DiRusso, Anthony M. DeMaria, Judy Wong…·Thomas D. Gilmore

This study identifies a conserved core region in NEMO essential for conformational changes and liquid-liquid phase separation in the NF-κB signaling pathway.

15 citations

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Molecular BiologyPLoS ONEPeer-reviewedSep 25, 2019

CRISPR/Cas9-based Editing of a Sensitive Transcriptional Regulatory Element to Achieve Cell Type-Specific Knockdown of the NEMO Scaffold Protein

Milad Babaei, Yuekun Liu, Shelly M. Wuerzberger‐Davis…·Thomas D. Gilmore

This paper uses CRISPR/Cas9 to disrupt a promoter element in 293T cells, eliminating NEMO expression and canonical NF-κB signaling while preserving non-canonical signaling.

10 citations

humanexperimental assay

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