Author

Soyoung Park

The University of Texas MD Anderson Cancer CenterORCID 0009-0005-0707-3426Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:metabolomics· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Daniela Rodriguez-Perera· 2 shared Ehsan IrajizadiD· 2 shared Fu Chung Hsiao· 2 shared Hiroyuki KatayamaiD· 2 shared Jennifer B. DennisoniD· 2 shared Jody Vykoukal· 2 shared Johannes Francois FahrmanniD· 2 shared Mingxin Zuo· 2 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

Daniela Rodriguez-Perera

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 6, 2026

KRAS-driven metabolic reprogramming of enhanced MFSD2A-mediated lysophosphatidylcholine scavenging promotes survival and progression of pancreatic ductal adenocarcinoma

Yihui Chen, Cason Haffner, Benson Chellakkan Selvanesan…·Johannes Francois Fahrmann

This study identifies KRAS-driven metabolic reprogramming through MFSD2A-mediated lysophosphatidylcholine scavenging as a driver of PDAC progression and shows that targeting this pathway with edelfosine has anti-cancer effects.

DOI 10.64898/2026.10.05.756824

humanbulk RNA-seq

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Cancer BiologyCell Reports MedicinePeer-reviewedDec 1, 2025

Vesicle-mediated mitochondrial clearance underlies an actionable metabolic vulnerability in triple-negative breast cancer

Jody Vykoukal, Yihui Chen, Mingxin Zuo…·Johannes Francois Fahrmann

The paper identifies a metabolic vulnerability in triple-negative breast cancer through sphingolipid metabolism, showing that targeting this pathway with eliglustat reduces tumor growth and improves survival.

6 citations

mouseproteomics

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GeneticseLifePeer-reviewedJan 15, 2018

p53 suppresses mutagenic RAD52 and POLθ pathways by orchestrating DNA replication restart homeostasis

Sunetra Roy, Karl-Heinz Tomaszowski, Jessica W. Luzwick…·Katharina Schlacher

This study identifies p53's role in suppressing mutagenic DNA repair pathways by regulating replication restart, revealing its importance in genomic stability and cancer therapy resistance.

87 citationstop 4%

humanmouse

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