Author

Laura M. Anchondo

University of California, RiversideORCID 0009-0007-8942-9768Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:statistical model· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Allison L. PhillipsiD· 2 shared Anthony E. BishayiD· 2 shared Bernhard Henkelmann· 2 shared Bhuvaneswari D. Chinthirla· 2 shared Duraan S. Olomi· 2 shared Eduardo Monarrez· 2 shared Elena V. KozlovaiD· 2 shared Gladys ChompréiD· 2 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Allison L. PhillipsORCID

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Author profile →

3 papers

Recent·Most cited

NeuroscienceJournal of NeuroendocrinologyPeer-reviewedAug 1, 2026

Enduring Autism-like Phenotypes and Deregulated Hypothalamic Prosocial Peptides After Early-Life Exposure to Indoor Flame Retardants in Male C57BL/6 Mice

Elena V. Kozlova, Gwendolyn Michelle Gonzalez, Maximillian E. Denys…·Margarita Currás‐Collazo

This study shows that early-life exposure to PBDEs in mice leads to autism-like behaviors and changes in prosocial neuropeptide expression.

DOI 10.1111/jne.70237

mousestatistical model

𝕏 PostBluesky

Developmental BiologyDiabetesPeer-reviewedJul 15, 2026

Developmental control of DNA damage responses in α- and β-cells shapes the selective beta-cell susceptibility in diabetes

Sneha S. Varghese, Alessandro Giovanni Hernandez-De La Peña, Liu Wang…·Sangeeta Dhawan

The paper investigates how developmental DNA damage responses in α- and β-cells contribute to their differential susceptibility to diabetes, showing β-cells have higher DNA damage vulnerability and repair efficiency.

DOI 10.2337/db25-1027

mouseDNA methylation

𝕏 PostBluesky

NeuroscienceArchives of ToxicologyPeer-reviewedOct 23, 2021

Persistent autism-relevant phenotype produced by in utero and lactational exposure of female mice to the commercial PBDE mixture, DE-71

Elena V. Kozlova, Matthew Christopher Valdez, Maximillian E. Denys…·Margarita Currás‐Collazo

This study shows that exposure to PBDE mixture DE-71 during pregnancy and lactation in female mice leads to autism-relevant behavioral and neurochemical changes in offspring.

32 citations

mousestatistical model

𝕏 PostBluesky