Author

Jenna A. Himelstein

Indiana University BloomingtonORCID 0009-0008-6486-4015Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:metabolomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Heather B. BradshawiD· 2 shared Taylor J. WoodwardiD· 2 shared Alexandros MakriyannisiD· 1 shared Andrea G. HohmanniD· 1 shared Carlos Henrique Alves JesusiD· 1 shared Éva WisniewskiiD· 1 shared Gergő SzandaiD· 1 shared John Hainline· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersNeuroscience

Heather B. BradshawORCID

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2 papers

Recent·Most cited

NeuroscienceNeuropharmacologyPeer-reviewedSep 7, 2026

Acetaminophen alters endogenous lipid signaling and attenuates pathological pain through a mechanism requiring diacylglycerol lipase, monoacylglycerol lipase and cannabinoid CB1 receptors in mice

Carlos Henrique Alves Jesus, Jonah L. Wirt, Taylor J. Woodward…·Andrea G. Hohmann

This study shows acetaminophen reduces pain by altering lipid signaling via cannabinoid receptors and enzymes in mice.

2 citations

mouseexperimental assay

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Molecular BiologybioRxivPreprintMay 5, 2026

Integrated lipidomic and transcriptomic analyses reveal novel endogenous lipid signaling system regulation in skin and plasma during psoriasiform inflammation

Éva Wisniewski, Wenwen Du, Jenna A. Himelstein…·Heather B. Bradshaw

The paper identifies compartment-specific changes in endolipid signaling during psoriasiform inflammation using lipidomic and transcriptomic analyses in mouse models.

DOI 10.64898/2026.05.01.722227

mousemetabolomics

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