Toxoplasma gondii associates with Benign Prostatic Hyperplasia and induces prostatic hyperplasia and urinary dysfunction in mice
Emily F. Stanczak, Tara D. Fuller, Douglas W. StrandiD, Hanyu XiaiD, Oliver R. StrobeliD, Irene Heredero‐BermejoiD, Gustavo ArrizabalagaiD, Travis J. JerdeiD
Abstract
ObjectivesBenign Prostatic Hyperplasia (BPH) is the non-cancerous enlargement of the prostate accompanied by lower urinary tract symptoms, affecting 50% of men by the age of 501,2. Advanced highly symptomatic BPH exhibits large epithelial glandular nodules with microglandular/atypical adenomatous hyperplasia, but how these features form is unknown3. Our lab has reported that the common parasite Toxoplasma gondii can infect the prostate and induce glandular nodule formation in mice3. The objective of this study is to determine if T. gondii exposure in humans correlates to BPH and nodule formation and if it induces urinary dysfunction concurrent in the mouse model. MethodsWe assessed Toxoplasma exposure by serum ELISA in patients with BPH and non-BPH donor controls, and compared seropositivity rates between the groups. We further assessed the histopathology of these patients for the presence of inflammation and epithelial glandular nodule formation and compared Toxoplasma positive and negative samples. We determined voiding function in Toxoplasma-infected mice between 14 and 60 days of infection with void spot with Void Whizzard software. ResultsMen diagnosed with BPH are more likely to be seropositive for Toxoplasma than age-matched undiagnosed donor controls. In addition, BPH patients that are seropositive for Toxoplasma are more likely to exhibit glandular nodule formation with microglandular / adenomous hyperplasia than seronegative BPH patients. In animal studies, Toxoplasma infection results in abnormal void patterns concurrent with microglandular hyperplasia and nodule formation. ConclusionsThese results suggest that Toxoplasma may be contributing to BPH pathology and lower urinary tract dysfunction in both humans and mice, opening new insights into the development of this important disease. The results also serve to further characterize this model of prostatic hyperplasia and define it as a potential urinary dysfunction model.
02 /Specification & Classification
Click any tag to browse matching papers- Citations
- 2
- Organism
- Approach
Reproducibility ledger
Nobody has reported trying to reproduce this paper yet. Did you replicate an experiment or run the code? Your report saves the next lab a week.
Sign in to file a reproducibility report.
Related papers
- CD8+ T CELLS ASSOCIATE WITH FORMING GLANDULAR NODULES IN TOXOPLASMA GONDII-INDUCED PROSTATIC HYPERPLASIA AND HUMAN BPH
Tara D. Fuller, Rafael B. Polidoro, … Travis J. Jerde·bioRxiv·Aug 20, 2026
- Presence of Toxoplasma gondii tissue cysts in human semen
W. H. Tong, J. Hlavacova, … A. Vyas·bioRxiv·Oct 27, 2021
- Steroid hormone imbalance drives macrophage infiltration and Spp1/osteopontin+ foam cell differentiation in the prostate
P. Popovics, K. O. Skalitzky, … W. A. Ricke·bioRxiv·Dec 13, 2022
- Prostatic osteopontin expression is associated with symptomatic benign prostatic hyperplasia
P. Popovics, W. N. Awadallah, … M. M. Grabowska·bioRxiv·Dec 23, 2019
- TNF Blockade Reduces Prostatic Hyperplasia and Inflammation while Limiting BPH Diagnosis in Patients with Autoimmune Disease
R. E. Vickman, L. Aaron-Brooks, … S. W. Hayward·bioRxiv·Mar 11, 2021
- Mast cell function in prostate inflammation, fibrosis, and smooth muscle cell dysfunction
G. Pattabiraman, A. J. Bell-Cohn, … P. Thumbikat·bioRxiv·Mar 23, 2021