Author

A. Babler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 2

Frequent Co-Authors

F. Gremse· 2 shared M. Herrmann· 2 shared W. Jahnen-Dechent· 2 shared A. Buescher· 1 shared C. Schmitz· 1 shared F. Kiessling· 1 shared I. Moshkova· 1 shared J. Floege· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

F. Gremse

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2 papers

Recent·Most cited

Microvasculopathy, Luminal Calcification and Premature Aging in Fetuin-A Deficient Mice

M. Herrmann, A. Babler, I. Moshkova, F. Gremse, F. Kiessling, U. Kusebauch, V. Nelea, R. Kramann, R. L. Moritz, M. D. McKee, W. Jahnen-Dechent

This study shows fetuin-A deficiency leads to microvascular calcification and premature aging in mice, associated with thrombosis and fibrosis rather than osteochondrogenic differentiation.

bioRxiv · Oct 4, 2019 · PreprintPhysiology

Microvasculopathy And Soft Tissue Calcification In Mice Are Governed by Fetuin-A, Pyrophosphate And Magnesium

A. Babler, C. Schmitz, A. Buescher, M. Herrmann, F. Gremse, T. G. Gorgels, J. Floege, W. Jahnen-Dechent

The paper identifies fetuin-A, pyrophosphate, and magnesium as key regulators of calcification in fetuin-A deficient mice and shows that their combined deficiency can be mitigated by dietary or parenteral supplementation.

bioRxiv · Oct 1, 2019Physiology