Author

A. Carminati

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

M. Deckert· 2 shared A. Baeri· 1 shared A. Lieu· 1 shared A. Prechtel· 1 shared A. Schnepf· 1 shared B. Mari· 1 shared C. Bontoux· 1 shared C. Girard· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

M. Deckert

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Author profile →

3 papers

Recent·Most cited

Disrupting USP39 deubiquitinase Function Impairs the Survival and Migration of Multiple Myeloma Cells through ZEB1 Degradation

J. Sirera, S. Sarlak, M. Teisseire, A. Carminati, C. Savy, V. Nicolini, P. Brest, C. Bontoux, M. Deckert, M. Ohanna, S. GIULIANO, M. Dufies, G. Pages, F. Luciano

This study identifies USP39 as a critical survival and migration factor in multiple myeloma cells, showing that its disruption leads to ZEB1 degradation and reduced tumor growth.

bioRxiv · Apr 23, 2024 · PreprintCancer Biology

Linking rhizosphere processes across scales: Opinion

A. Schnepf, A. Carminati, M. Ahmed, M. Ani, P. Benard, J. Bentz, M. Bonkowski, M. Brax, D. Diehl, P. Duddek, E. Kroener, M. Javaux, M. Landl, E. Lehndorff, E. Lippold, , D. Vetterlein

The paper discusses how multi-scale rhizosphere processes influence plant-scale behavior and presents models that integrate experimental data across scales to predict rhizodeposition and microbial activity.

bioRxiv · Jul 8, 2021Bioengineering

Blockade of pro-fibrotic response mediated by the miR-143/-145 cluster prevents targeted therapy-induced phenotypic plasticity and resistance in melanoma

S. Diazzi, A. Baeri, J. Fassy, M. Lecacheur, O. Marin-Bejar, C. Girard, L. Lefevre, C. Lacoux, M. Irondelle, C. Mounier, M. Truchi, M. Couralet, A. Carminati, I. Berestjuk, F. Larbret, , S. Tartare-Deckert

This study identifies the miR-143/-145 cluster as a driver of therapy-resistant mesenchymal-like phenotype in melanoma and shows that blocking its pro-fibrotic effects enhances MAPK inhibitor efficacy.

bioRxiv · Jul 2, 2021Cancer Biology