Author

M. Truchi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Assay:scRNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1

Frequent Co-Authors

B. Mari· 4 shared A. Baeri· 2 shared A. Lingampally· 2 shared G. Vassaux· 2 shared J. Fassy· 2 shared S. Bellusci· 2 shared A. Carminati· 1 shared A. Daher· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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4 shared papersPathology

B. Mari

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Lineage-tracing of Acta2+ cells in aged mice during lung fibrosis formation and resolution supports the lipofibroblasts to myofibroblast reversible switch

A. Lingampally, M. Truchi, O. Mauduit, V. Delcroix, S. Hadzic, J. Koepke, A.-I. Vazquez-Armendariz, S. Herold, C. Samakovlis, H. Makarenkova, E. El Agha, C. Chen, B. Mari, S. Bellusci

This study traces Acta2+ cells in aged mice during lung fibrosis to show lipofibroblasts can reversibly switch to myofibroblasts, with similar patterns observed in humans.

bioRxiv · Feb 19, 2024 · PreprintPathology

Aging affects reprogramming of murine pulmonary capillary endothelial cells after lung injury

M. Truchi, M. Gautier-Isola, G. Savary, H. Cadis, C. Scribe, A. Baeri, A. Lingampally, V. Magnone, C. Girard-Riboulleau, M.-J. Arguel, C. de Schutter, J. Fassy, N. Boukrout, R. Larrue, N. Martin, , B. Mari

This study compares transcriptomic changes in pulmonary capillary endothelial cells from young and aged mice after lung injury, finding age-related differences in resolution and fibrotic signatures.

bioRxiv · Jul 11, 2023 · PreprintPathology

The sequestration of miR-642a-3p by a complex formed by HIV-1 Gag and human Dicer increases AFF4 expression and viral production

S. P. Alpuche-Lazcano, O. R. Dunkley, R. J. Scarborough, S. M. Daniels, A. Daher, M. Truchi, M. C. Estable, B. Mari, A. J. Mouland, A. Gatignol

The paper shows HIV-1 Gag sequesters miR-642a-3p with Dicer, increasing AFF4 expression and HIV-1 production by disrupting miR-642a-3p's antiviral activity.

bioRxiv · May 24, 2023 · PreprintMicrobiology

Blockade of pro-fibrotic response mediated by the miR-143/-145 cluster prevents targeted therapy-induced phenotypic plasticity and resistance in melanoma

S. Diazzi, A. Baeri, J. Fassy, M. Lecacheur, O. Marin-Bejar, C. Girard, L. Lefevre, C. Lacoux, M. Irondelle, C. Mounier, M. Truchi, M. Couralet, A. Carminati, I. Berestjuk, F. Larbret, , S. Tartare-Deckert

This study identifies the miR-143/-145 cluster as a driver of therapy-resistant mesenchymal-like phenotype in melanoma and shows that blocking its pro-fibrotic effects enhances MAPK inhibitor efficacy.

bioRxiv · Jul 2, 2021Cancer Biology