Author

A. E. Greenberg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

M. J. Greenberg· 2 shared A. L. Bredemeyer· 1 shared B. Garcia· 1 shared G. Meyer· 1 shared J. A. Sanford· 1 shared J. L. A. Ferey· 1 shared K. Lavine· 1 shared K. Svensson· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiophysics

M. J. Greenberg

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Author profile →

2 papers

Recent·Most cited

Harnessing molecular mechanism for precision medicine in dilated cardiomyopathy caused by a mutation in troponin T

L. Greenberg, W. T. Stump, Z. J. Lin, A. L. Bredemeyer, T. Blackwell, X. Hao, A. E. Greenberg, B. Garcia, K. Lavine, M. J. Greenberg

This study connects genotype, phenotype, and drug response in dilated cardiomyopathy using molecular mechanism-based computational and experimental approaches for a troponin T mutation.

bioRxiv · Apr 5, 2024 · PreprintBiophysics

Insights into post-translational regulation of skeletal muscle contractile function by the acetyltransferases, p300 and CBP

G. Meyer, J. L. A. Ferey, J. A. Sanford, L. S. Fitzgerald, A. E. Greenberg, K. Svensson, M. J. Greenberg, S. Schenk

This study shows that loss of p300 and CBP in skeletal muscle causes severe contractile dysfunction, but mitochondrial function and cross-bridge cycling are not the primary causes.

bioRxiv · Feb 27, 2024Cell Biology