A. K. Criss

Neisseria gonorrhoeae scavenges host sialic acid for Siglec-mediated, complement-independent suppression of neutrophil activation

This paper shows that Neisseria gonorrhoeae uses sialic acid from the host to modify its surface, which helps it avoid neutrophil activation without needing the complement system.

Evaluating vaccine-elicited antibody activities against Neisseria gonorrhoeae: cross-protective responses elicited by the 4CMenB meningococcal vaccine

This study evaluates antibody activities against Neisseria gonorrhoeae using assays to assess vaccine efficacy, showing cross-protective responses from the 4CMenB meningococcal vaccine.

Transcriptome guided metabolic network analysis reveals rearrangements of carbon flux distribution in Neisseria gonorrhoeae during neutrophil co-culture

This paper uses transcriptome data and metabolic modeling to show Neisseria gonorrhoeae reorganizes its carbon flux during co-culture with neutrophils, enhancing its survival.

Neisseria gonorrhoeae co-opts C4b-binding protein to enhance complement-independent survival from neutrophils

This study shows that Neisseria gonorrhoeae uses C4b-binding protein to survive neutrophil attacks without complement involvement, enhancing its persistence in human mucosal environments.

Phagocytosis via complement receptor 3 enables microbes to evade killing by neutrophils

This study shows that CR3-mediated phagocytosis allows pathogens to avoid neutrophil killing by suppressing ROS production and forming immature phagosomes.