Author

A. L. Armijo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:chromatin & epigenetic regulation· 2 Assay:DNA methylation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:whole-genome sequencing· 1

Frequent Co-Authors

B. P. Engelward· 2 shared J. J. Corrigan· 2 shared J. M. Essigmann· 2 shared R. G. Croy· 2 shared A. F. Dormitzer· 1 shared B. I. Fedeles· 1 shared D. K. Torous· 1 shared D. L. Plata· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPharmacology & Toxicology

B. P. Engelward

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

DNA adduct and mutational profiles reveal a threshold of cellular defenses against N-nitrosodimethylamine administered to mice in drinking water

N. E. Gubina, L. B. Volk, A. F. Dormitzer, E. M. Michelsen, L. J. Pribyl, J. J. Corrigan, E. D. Dalvie, A. L. Armijo, M. M. Norales, N. A. Bugher, K. M. Schonvisky, D. L. Plata, B. P. Engelward, R. G. Croy, J. M. Essigmann, B. I. Fedeles

This study examines DNA adducts and mutations in mice exposed to NDMA, finding that m6G adducts and mutations increase beyond a certain dose threshold, implicating MGMT saturation in mutagenesis.

bioRxiv · Nov 13, 2025 · PreprintPharmacology & Toxicology

Excision of mutagenic replication-blocking lesions suppresses cancer but promotes cytotoxicity and lethality in nitrosamine-exposed mice

J. E. Kay, J. J. Corrigan, A. L. Armijo, I. S. Nazari, I. N. Kohale, D. K. Torous, S. L. Avlasevich, R. G. Croy, D. N. Wadduwage, S. E. Carrasco, S. D. Dertinger, F. M. White, J. M. Essigmann, L. D. Samson, B. P. Engelward

The paper shows that altering AAG levels in mice changes the outcome of NDMA exposure from cancer to lethality by shifting the balance between replication-blocking lesions and strand breaks.

bioRxiv · Jan 12, 2021Cancer Biology