Author

J. E. Kay

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

A. L. Armijo· 1 shared B. P. Engelward· 1 shared D. K. Torous· 1 shared D. N. Wadduwage· 1 shared F. M. White· 1 shared I. N. Kohale· 1 shared I. S. Nazari· 1 shared J. J. Corrigan· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. L. Armijo

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Excision of mutagenic replication-blocking lesions suppresses cancer but promotes cytotoxicity and lethality in nitrosamine-exposed mice

J. E. Kay, J. J. Corrigan, A. L. Armijo, I. S. Nazari, I. N. Kohale, D. K. Torous, S. L. Avlasevich, R. G. Croy, D. N. Wadduwage, S. E. Carrasco, S. D. Dertinger, F. M. White, J. M. Essigmann, L. D. Samson, B. P. Engelward

The paper shows that altering AAG levels in mice changes the outcome of NDMA exposure from cancer to lethality by shifting the balance between replication-blocking lesions and strand breaks.

bioRxiv · Jan 12, 2021Cancer Biology