Author

A. M. Duncan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Organism:mouse· 1 Organism:zebrafish· 1

Frequent Co-Authors

M. A. Brindley· 3 shared M. D. Acciani· 3 shared A. R. Jimenez· 2 shared J. M. Reyes Ballista· 2 shared K. E. Havranek· 2 shared K. L. Miazgowicz· 2 shared A. J. Hoover· 1 shared D. N. Barnes· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersMicrobiology

M. A. Brindley

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Chikungunya Virus Release is Reduced by TIM-1 Receptors Through Binding of Envelope Phosphatidylserine

J. M. Reyes Ballista, A. J. Hoover, J. T. Noble, M. D. Acciani, K. L. Miazgowicz, S. A. Harrison, G. A. L. Tabscott, A. M. Duncan, D. N. Barnes, A. R. Jimenez, M. A. Brindley

This study shows TIM-1 receptors reduce Chikungunya virus release by binding envelope phosphatidylserine, with virus counteracting this by decreasing TIM-1 surface levels.

Journal of Virology · Aug 20, 2024Peer-reviewedMicrobiology

Phosphatidylserine within the Viral Membrane Enhances Chikungunya Virus Infectivity in a Cell-type Dependent Manner

K. L. Miazgowicz, J. M. Reyes Ballista, M. D. Acciani, A. R. Jimenez, R. S. Belloli, A. M. Duncan, K. E. Havranek, M. A. Brindley

This study shows that Chikungunya virus infectivity depends on phosphatidylserine levels in its membrane and is cell-type dependent.

bioRxiv · Jan 14, 2022 · PreprintMicrobiology

Ebola virus requires phosphatidylserine scrambling activity for efficient budding and optimal infectivity

M. D. Acciani, M. F. Lay-Mendoza, K. E. Havranek, O. L. Linn, A. M. Duncan, H. Iyer, M. A. Brindley

This study shows that XKR8, not TMEM16F, is essential for Ebola virus budding and infectivity by enabling phosphatidylserine (PS) externalization on the viral envelope.

bioRxiv · Mar 16, 2020Microbiology