Author

A. Pfenniger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Physiology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 1 Assay:electrophysiology· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Burrell· 2 shared B. P. Knight· 2 shared D. A. Johnson· 2 shared J. A. Wasserstrom· 2 shared J. Ng· 2 shared R. Arora· 2 shared R. Passman· 2 shared S. Yoo· 2 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

A. Burrell

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Regions of Highly Recurrent Electrogram Morphology at Sites of Low Cycle Length Accurately Reflect Arrhythmogenic Substrate for Atrial Fibrillation - Implications For a New, Mechanism Guided Therapeutic Approach for Atrial Fibrillation

S. Yoo, M. Rottmann, J. Ng, D. A. Johnson, B. M. Shanab, A. Pfenniger, G. E. Geist, S. Mandava, A. Burrell, W. Zhang, J. A. Wasserstrom, B. P. Knight, R. Passman, J. Goldberger, R. Arora

This study identifies electrogram morphology recurrence as a marker of arrhythmogenic substrate in atrial fibrillation, suggesting it could guide targeted ablation therapy.

bioRxiv · Dec 10, 2020 · PreprintPhysiology

Disruption of NOX2-dependent Oxidative Injury with a Targeted Gene-Therapy Approach Prevents Atrial Fibrillation in a Canine Model

S. Yoo, A. Pfenniger, J. Hoffman, W. Zhang, J. Ng, A. Burrell, D. A. Johnson, G. Gussak, T. Waugh, S. Bull, B. Benefield, B. P. Knight, R. Passman, J. A. Wasserstrom, G. L. Aistrup, R. Arora

This study shows that disrupting NOX2-dependent oxidative injury with gene therapy prevents atrial fibrillation in dogs.

bioRxiv · Sep 11, 2019 · PreprintPhysiology