Author

A. R. Kroken

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Assay:proteomics· 1

Frequent Co-Authors

D. J. Evans· 2 shared S. M. J. Fleiszig· 2 shared V. Nieto· 2 shared E. J. Jedel· 1 shared E. Jedel· 1 shared K. A. Klein· 1 shared M. E. Hallsten· 1 shared M. M. E. Metruccio· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersMicrobiology

D. J. Evans

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Intracellular replication of Pseudomonas aeruginosa in epithelial cells requires suppression of the caspase-4 inflammasome

A. R. Kroken, K. A. Klein, P. S. Mitchell, V. Nieto, E. J. Jedel, D. J. Evans, S. M. J. Fleiszig

This study shows that Pseudomonas aeruginosa suppresses the caspase-4 inflammasome to enable intracellular replication in epithelial cells, highlighting a new role for ExoS in inhibiting host cell death.

bioRxiv · Feb 13, 2023 · PreprintMicrobiology

Pseudomonas aeruginosa can diversify after host cell invasion to establish multiple intracellular niches

N. G. Kumar, V. Nieto, A. R. Kroken, E. Jedel, M. R. Grosser, M. E. Hallsten, M. M. E. Metruccio, T. L. Yahr, D. J. Evans, S. M. J. Fleiszig

This study shows Pseudomonas aeruginosa can persist in vacuoles within host cells despite lacking the T3SS, tolerating antibiotics, and contributing to pathogenesis through intracellular diversification.

bioRxiv · Oct 7, 2022Microbiology