Author

A. R. Sullivan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Ecology· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1 Assay:DNA methylation· 1 Research Focus:ecology & biodiversity· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:metagenomics· 1

Frequent Co-Authors

A. B. Siddique· 1 shared A. D. Drouzas· 1 shared A. L. Curtu· 1 shared A. Mikko· 1 shared A. Norman· 1 shared A. Sjodin· 1 shared A.-M. Johansson· 1 shared A.-S. Wozny· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperEcology

A. B. Siddique

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Airborne eDNA captures three decades of ecosystem biodiversity

A. R. Sullivan, E. Karlsson, D. Svensson, B. Brindefalk, J. Antonio Villegas, A. Mikko, D. Bellieny, A. B. Siddique, A.-M. Johansson, H. Grahn, D. Sundell, A. Norman, P.-A. Esseen, A. Sjodin, N. J. Singh, , P. Stenberg

The paper develops a method using airborne eDNA sequencing to monitor biodiversity over 34 years, detecting over 2,700 genera and revealing regional biodiversity declines.

bioRxiv · Dec 6, 2023Ecology

Scots pine - panmixia and the elusive signal of genetic adaptation

J. Bruxaux, W. Zhao, D. Hall, A. L. Curtu, P. Androsiuk, A. D. Drouzas, O. Gailing, H. Konrad, A. R. Sullivan, V. Semerikov, X.-R. Wang

This study analyzes genetic diversity and adaptation in Scots pine across Eurasia, finding high genetic uniformity and limited signals of adaptation.

bioRxiv · Jun 9, 2023Evolutionary Biology

Hyperactive end joining repair mediates resistance to DNA damaging therapy in p53-deficient cells

R. J. Kumar, H. X. Chao, V. R. Roberts, A. R. Sullivan, S. J. Shah, D. A. Simpson, W. Feng, A.-S. Wozny, S. Kumar, J. E. Purvis, G. P. Gupta

The paper identifies hyperactive end joining repair in p53-deficient cells as a mechanism of resistance to DNA-damaging therapies and shows that combined inhibition of DNA-PK and Pol theta restores sensitivity.

bioRxiv · Apr 1, 2020 · PreprintCell Biology