Author

A. Stender

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:metagenomics· 1 Organism:mouse· 1

Frequent Co-Authors

J. Li· 1 shared L. Klemke· 1 shared N. Stark· 1 shared O. C. Sener· 1 shared R. Schulz-Heddergott· 1 shared T. Isermann· 1 shared U. M. Moll· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

J. Li

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1 paper

Recent·Most cited

Suppression of HSF1 activity by wildtype p53 creates the driving force for p53 loss-of-heterozygosity, enabling mutant p53 stabilization and invasion

O. C. Sener, A. Stender, L. Klemke, N. Stark, T. Isermann, J. Li, U. M. Moll, R. Schulz-Heddergott

This paper identifies the WTp53-HSF1 axis as the driver of p53LOH in colorectal tumors, enabling mutp53 stabilization and invasion.

bioRxiv · Apr 23, 2020Cancer Biology