Author

Ahmed Ismail

Cold Spring Harbor LaboratoryPrimary ORCID profile (0000-0002-0104-6540 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Ahmed IsmailiD1 paper

Biology Disciplines

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Developmental Biology· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:bacteria (other)· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Alison J. SeboldiD· 1 shared Andrew Garcia· 1 shared Bin ZhuiD· 1 shared Cheolju LeeiD· 1 shared David Bolton· 1 shared Elaine J. Marchi· 1 shared Gholson J. LyoniD· 1 shared Goo Taeg OhiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperDevelopmental Biology

Alison J. SeboldORCID

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2 papers

Recent·Most cited

Experimental evolution of gene essentiality in bacteria

Liang Bao, Zan Zhu, Ahmed Ismail, Bin Zhu, Vysakh Anandan, Marvin Whiteley, Todd O. Kitten, Ping Xu

The paper generates essential gene deletion mutants in Streptococcus sanguinis and identifies suppressor mutations through experimental evolution, revealing new gene and pathway relationships.

mBio · Oct 31, 2025Peer-reviewedEvolutionary Biology

Naa12 compensates for Naa10 in mice in the amino-terminal acetylation pathway

Hyae Yon Kweon, Mi-Ni Lee, Max Johannes Dörfel, Seungwoon Seo, Leah Gottlieb, Thomas Papazyan, Nina McTiernan, Rasmus Ree, David Bolton, Andrew Garcia, Michael J. Flory, Jonathan Crain, Alison J. Sebold, Scott K. Lyons, Ahmed Ismail, , Gholson J. Lyon

The paper identifies Naa12 as a compensatory N-terminal acetyltransferase that rescues Naa10 deficiency in mice, revealing its role in amino-terminal acetylation.

eLife · Aug 6, 2021Peer-reviewedDevelopmental Biology