Author

Anthony M. Downs

Emory UniversityPrimary ORCID profile (0000-0003-0146-4459 · 6 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Anthony M. DownsiD6 papers · University of North Carolina at Chapel Hill

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Ellen J. HessiD· 2 shared Hyder Azad JinnahiD· 2 shared Xueliang Fan· 2 shared Yuping DonsanteiD· 2 shared Ashok Reddy DinasarapuiD· 1 shared Jessica HoehneriD· 1 shared Kaitlyn M. Roman· 1 shared Maria A. Briscione· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Ellen J. HessORCID

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2 papers

Recent·Most cited

Cell-intrinsic effects of TorsinA(ΔE) disrupt dopamine release in a mouse model of DYT1-TOR1A dystonia

Anthony M. Downs, Xueliang Fan, Radhika F. Kadakia, Yuping Donsante, Hyder Azad Jinnah, Ellen J. Hess

This study shows that the Tor1a(ΔE) mutation in dopamine neurons reduces dopamine release, while having no effect in cholinergic neurons, indicating a cell-intrinsic role in dystonia.

Neurobiology of Disease · Apr 21, 2021Peer-reviewedNeuroscience

Differential expression of striatal proteins in a mouse model of DOPA-responsive dystonia reveals shared mechanisms among dystonic disorders

Maria A. Briscione, Ashok Reddy Dinasarapu, Pritha Bagchi, Yuping Donsante, Kaitlyn M. Roman, Anthony M. Downs, Xueliang Fan, Jessica Hoehner, Hyder Azad Jinnah, Ellen J. Hess

The paper identifies differentially expressed proteins in a mouse model of DOPA-responsive dystonia, revealing shared mechanisms with human dystonic disorders.

bioRxiv · Jan 12, 2021Neuroscience