Author

Aras N. Mattis

Primary ORCID profile (0000-0002-5813-164X · 5 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Aras N. MattisiD5 papers · University of California, San Francisco

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

Camella G. Wilson· 1 shared Ethan J. Weiss· 1 shared Jennifer L. Tran· 1 shared Kevin Corbit· 1 shared

Activity by Year

2016-2016 · peak 1/yr

Co-Author Collaboration Network

4 top collaborators · 6 mutual co-author links

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1 shared paperPhysiology

Camella G. Wilson

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1 paper

Recent·Most cited

Disruption of Hepatocyte Jak2 leads to Spontaneous NASH in Aged Mice and Uncouples Metabolic Liver Disease from Insulin Resistance

Camella G. Wilson, Aras N. Mattis, Jennifer L. Tran, Kevin Corbit, Ethan J. Weiss

This paper shows that disrupting Jak2 in hepatocytes causes spontaneous NASH in aged mice without dietary changes, while adipocyte Jak2 loss improves insulin sensitivity.

bioRxiv · Oct 4, 2016 · PreprintPhysiology