Author

B. E. Fee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Assay:proteomics· 1

Frequent Co-Authors

G. E. Hammer· 2 shared H.-I. Huang· 2 shared N. Youssef· 2 shared A. F. Buckley· 1 shared A. R. Rogala· 1 shared A. S. Gulati· 1 shared A. Schoenborn· 1 shared B. A. Vallance· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersImmunology

G. E. Hammer

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2 papers

Recent·Most cited

Th17 immunity in the colon is controlled by two novel subsets of colon-specific mononuclear phagocytes

H.-I. Huang, M. L. Jewell, N. Youssef, M.-N. Huang, E. Hauser, B. E. Fee, N. P. Rudemiller, J. Privratsky, J. J. Zhang, E. Reyes, D. Wang, G. A. Taylor, M. D. Gunn, D. C. Ko, D. N. Cook, , G. E. Hammer

This study identifies two novel colon-specific mononuclear phagocyte subsets that are critical for Th17 immunity in the colon.

bioRxiv · Jan 28, 2021Immunology

Irgm1-deficiency leads to myeloid dysfunction in colon lamina propria and susceptibility to the intestinal pathogen Citrobacter rodentium

G. Taylor, H.-I. Huang, B. E. Fee, N. Youssef, V. Cantillana, A. Schoenborn, A. R. Rogala, A. F. Buckley, B. A. Vallance, A. S. Gulati, G. E. Hammer

This study shows that Irgm1 deficiency causes myeloid cell dysfunction in the colon and increased susceptibility to Citrobacter rodentium infection, independent of intracellular killing.

bioRxiv · Mar 26, 2019Immunology