Author

B. E. Flucher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Assay:electrophysiology· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1

Frequent Co-Authors

K. R. Liedl· 2 shared A. L. Kochiss· 1 shared D. Wilflingseder· 1 shared H. J. Draheim· 1 shared J. Striessnig· 1 shared M. Ben-Johny· 1 shared M. Campiglio· 1 shared M. L. Fernandez-Quintero· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBiophysics

K. R. Liedl

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2 papers

Recent·Most cited

Asymmetric Contribution of a Selectivity Filter Gate in Triggering Inactivation of CaV1.3 Channels

P. J. del Rivero Morfin, A. L. Kochiss, K. R. Liedl, B. E. Flucher, M. L. I. Fernandez-Quintero, M. Ben-Johny

The paper investigates how asymmetric conformational changes in CaV1.3 channels mediate voltage- and calcium-dependent inactivation through distinct molecular mechanisms.

bioRxiv · Sep 21, 2023Biophysics

Calcium current modulation by the γ1 subunit depends on alternative splicing of CaV1.1

Y. E. El Ghaleb, N. J. Ortner, W. Posch, M. L. Fernandez-Quintero, W. E. Tuinte, S. Monteleone, H. J. Draheim, K. R. Liedl, D. Wilflingseder, J. Striessnig, P. Tuluc, B. E. Flucher, M. Campiglio

This study shows that the γ1 subunit reduces current density of CaV1.1a but not CaV1.1e, and this effect depends on alternative splicing of exon 29 in CaV1.1a.

bioRxiv · Nov 10, 2021 · PreprintPhysiology