Author

B. Holzmann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Bleich· 1 shared A. Garcias Lopez· 1 shared A. Sichler· 1 shared A. Sitnow· 1 shared B. Malissen· 1 shared C. Mogler· 1 shared C. Ratiu· 1 shared D. Hartmann· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCell Biology

A. Bleich

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The gut microbiota promotes liver regeneration through hepatic membrane phospholipid synthesis

Y. Yin, A. Sichler, J. Ecker, M. Laschinger, G. Liebisch, M. Hoering, M. Basic, A. Bleich, X.-J. Zhang, P. Stupakov, Y. Gaertner, F. Lohoefer, C. Mogler, H. Friess, D. Hartmann, , K.-P. Janssen

This study shows that gut microbiota supports liver regeneration by promoting hepatic phospholipid synthesis, with antibiotic-induced dysbiosis impairing this process.

bioRxiv · Aug 25, 2022 · PreprintCell Biology

Notch and TLR signaling coordinate monocyte cell fate and inflammation

J. Gamrekelashvili, T. Kapanadze, S. Sablotny, C. Ratiu, K. Dastagir, M. Lochner, S. Karbacher, P. Wenzel, A. Sitnow, S. Fleig, T. Sparwasser, U. Kalinke, H. Haller, B. Holzmann, F. Limbourg

This study shows that Notch2 and TLR7-Myd88 pathways regulate Ly6Chi monocyte differentiation and inflammation in mice.

bioRxiv · Apr 28, 2020Immunology

Migration of intestinal dendritic cell subsets upon intrinsic and extrinsic TLR3 stimulation

A. Garcias Lopez, V. Bekiaris, K. Müller Luda, J. Hütter, K. Getachew Muleta, J. Nakawesi, I. Ulmert, K. Kotarsky, B. Malissen, M. O'Keeffe, B. Holzmann, W. W. Agace, K. Lahl

This study shows that cDC1 and cDC2 subsets migrate similarly in response to TLR3 stimulation, but cDC1 requires type I interferon signaling, highlighting distinct pathways in DC subset migration.

bioRxiv · Nov 14, 2019Immunology