Author

B. Plougastel-Douglas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

B. A. Parikh· 1 shared D. L. Beckman· 1 shared E. Oltz· 1 shared E. Park· 1 shared J. Poursine-Laurent· 1 shared K. M. Murphy· 1 shared K. Zaitsev· 1 shared L. D. Sibley· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

B. A. Parikh

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2 papers

Recent·Most cited

Major histocompatibility complex class I-restricted protection against murine cytomegalovirus requires missing-self recognition by the natural killer cell inhibitory Ly49 receptors

B. A. Parikh, M. D. Bern, S. J. Piersma, L. Yang, D. L. Beckman, J. Poursine-Laurent, B. Plougastel-Douglas, W. M. M. Yokoyama

This study shows that NK cell inhibitory Ly49 receptors are essential for controlling MCMV infection in an MHC-restricted manner, supporting the missing-self hypothesis.

bioRxiv · Sep 4, 2019 · PreprintImmunology

Toxoplasma gondii infection drives conversion of NK Cells into ILC1s

E. Park, S. Patel, Q. Wang, P. Andhey, K. Zaitsev, S. Porter, M. Hershey, M. Bern, B. Plougastel-Douglas, P. Collins, M. Colonna, K. M. Murphy, E. Oltz, M. Artyomov, L. D. Sibley, W. M. Yokoyama

This study shows Toxoplasma gondii infection converts NK cells into ILC1-like cells that persist and circulate independently of infection.

bioRxiv · May 17, 2019Immunology