Author

C. Bonfanti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. M. Valenza· 1 shared G. Messina· 1 shared G. Temponi· 1 shared J. Pollard· 1 shared L. Arnaboldi· 1 shared M. Cassinelli· 1 shared M. E. Pasini· 1 shared M. Frattaroli· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

A. M. Valenza

Mutual co-authors in cluster

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2 papers

Recent·Most cited

A Novel Drosophila Model to Investigate Adipose Tissue Macrophage Infiltration (ATM) and Obesity highlights the Therapeutic Potential of Attenuating Eiger/TNF Signaling to Ameliorate Insulin Resistance and ATM.

Z. Mirzoyan, A. M. Valenza, S. Zola, C. Bonfanti, L. Arnaboldi, N. Ferrari, J. Pollard, V. Lupi, M. Cassinelli, M. Frattaroli, M. Sahin, M. E. Pasini, P. Bellosta

This study develops a Drosophila model to investigate adipose tissue macrophage infiltration and obesity, showing that reducing Eiger/TNF signaling improves insulin resistance and reduces immune cell infiltration.

bioRxiv · Jul 6, 2023Physiology

Selective ablation of Nfix in Macrophages preserves Muscular Dystrophy by inhibiting FAPs-dependent fibrosis

M. Saclier, G. Temponi, C. Bonfanti, G. Messina

This study shows that inhibiting Nfix in macrophages reduces fibrosis in muscular dystrophy models by decreasing fibro-adipogenic progenitor survival.

bioRxiv · May 12, 2021Cell Biology