Author

C. Delannoy

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1 Zoology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Assay:proteomics· 3 Organism:human· 1 Organism:mouse· 1 Organism:non-human primate· 1

Frequent Co-Authors

Y. Guerardel· 3 shared A. Di Maio· 1 shared C. Bressollette-Bodin· 1 shared C. C. Feral· 1 shared C. Iltis· 1 shared C. Sato· 1 shared D. Ebert· 1 shared D. McILROY· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersZoology

Y. Guerardel

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Anemonefish use sialic acid metabolism as Trojan horse to avoid giant sea anemone stinging

N. Roux, C. Delannoy, S.-Y. Yu, S. Miura, L. Carlu, L. Besseau, T. Nakagawa, C. Sato, K. Kitajima, Y. Guerardel, V. Laudet

This paper investigates how anemonefish avoid triggering sea anemone nematocysts by reducing sialic acid levels in their mucus, suggesting a Trojan horse mechanism for symbiosis.

bioRxiv · Apr 22, 2024Zoology

Structural and functional analysis of natural capsid variants reveals sialic-acid independent entry of BK polyomavirus

M. Sorin, A. Di Maio, L. M. Silva, D. Ebert, C. Delannoy, N.-K. Nguyen, Y. Guerardel, W. Chai, F. Halary, K. Renaudin-Autain, Y. Liu, C. Bressollette-Bodin, T. Stehle, D. McILROY

This study analyzes natural VP1 mutations in BKPyV to show some variants can enter cells without sialic acid, suggesting an alternative receptor.

bioRxiv · Jul 13, 2022 · PreprintMicrobiology

A ganglioside-based senescence-associated immune checkpoint

C. Iltis, L. Seguin, L. Cervera, L. Duret, T. Hamidouch, S. Kunz, O. Croce, C. Delannoy, Y. Guerardel, F. Allain, L. Moudombi, P. Hofman, E. Cosson, J. Guglielmi, T. Pourcher, , J. Cherfils-Vicini

This paper identifies GD3 as a senescence-associated immune checkpoint that suppresses NK cell-mediated clearance of senescent cells, showing that targeting GD3 can enhance anti-fibrotic effects in a mouse model.

bioRxiv · Apr 23, 2021 · PreprintImmunology