Author

C. J. Buchholz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Braun· 1 shared A. Jamali· 1 shared A. Michels· 1 shared E. Adabi· 1 shared E. Flory· 1 shared F. Thalheimer· 1 shared K. Cichutek· 1 shared N. Ho· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperPharmacology & Toxicology

A. Braun

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Early induction of severe cytokine release syndrome by rapidly generated CAR T cells in a preclinical mouse model

N. Ho, A. Jamali, A. Braun, E. Adabi, F. Thalheimer, C. J. Buchholz

This study shows that rapidly generated CAR T cells induce severe cytokine release syndrome in a mouse model within 24 hours.

bioRxiv · Nov 12, 2022 · PreprintPharmacology & Toxicology

Quantitative Assays Reveal Cell Fusion at Minimal Levels of SARS-CoV-2 Spike Protein and Fusion-from-Without

S. A. Theuerkauf, A. Michels, V. Riechert, T. J. Maier, E. Flory, K. Cichutek, C. J. Buchholz

The paper develops quantitative assays to study SARS-CoV-2 spike protein-mediated cell fusion, revealing that syncytia formation can occur at low S protein levels and is less inhibited by antibodies than particle-cell fusion.

bioRxiv · Oct 15, 2020Microbiology