Acute kidney injury-induced circulating TNFR1/2 elevations correlate with persistent kidney injury and progression to fibrosis
A. Arthanarisami, Y. Komaru, C. Katsouridi, J. Schumacher, D. K. Verges, L. Ning, M. M. Abdelmageed, A. Herrlich, E. Kefaloyianni
This study shows that acute kidney injury (AKI) increases circulating TNFR1/2 levels, which correlate with kidney injury severity and progression to fibrosis, particularly through extracellular vesicle-bound forms.
Cells · Sep 5, 2023Peer-reviewedPhysiology