Author

C. M. Haynes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

C. Wang· 1 shared D. Chen· 1 shared F. Zhang· 1 shared H. Liu· 1 shared J.-H. Shim· 1 shared K. W. Nehrke· 1 shared L. Zhang· 1 shared M. N. McCall· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

C. Wang

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2 papers

Recent·Most cited

The ESCRT protein CHMP5 restricts bone formation by controlling endolysosome-mitochondrion-mediated cell senescence

F. Zhang, Y. Wang, L. Zhang, C. Wang, D. Chen, H. Liu, R. Xu, C. M. Haynes, J.-H. Shim, X. Ge

This study shows that CHMP5 regulates bone formation by controlling endolysosome-mitochondrion-mediated cell senescence, and targeting senescent cells can alleviate musculoskeletal abnormalities.

bioRxiv · Aug 3, 2020Cell Biology

Cardioprotection by the mitochondrial unfolded protein response (UPRmt) is mediated by activating transcription factor 5 (ATF5)

Y. T. Wang, Y. Lim, M. N. McCall, C. M. Haynes, K. W. Nehrke, P. S. Brookes

This study shows that ATF5 is required for cardioprotection induced by UPRmt activators against ischemia-reperfusion injury.

bioRxiv · Nov 9, 2018Cell Biology