Author

C. M. P. Sabusap

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Animal Behavior & Cognition· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

L. Plate· 3 shared E. F. McDonald· 2 shared M. Kim· 2 shared A. G. Palacios· 1 shared A. O. Ardiles· 1 shared A. Rab· 1 shared B. Morales· 1 shared B. Timalsina· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersCell Biology

L. Plate

Mutual co-authors in cluster

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3 papers

Recent·Most cited

The unfolded protein response transcription factor XBP1s ameliorates Alzheimer`s disease by improving synaptic function and proteostasis

C. Duran Aniotz, C. Rivera Krstulovic, N. Poblete, A. O. Ardiles, M. L. Diaz Hung, C. M. P. Sabusap, Y. Gerakis, F. Cabral Miranda, J. Diaz, M. Fuentealba, E. Munoz, S. Espinosa, G. Matinez, G. Quiroz, G. Tamburini, , C. Hetz

This study shows that overexpressing the active form of XBP1 in neurons reduces amyloid deposits and improves synaptic function in Alzheimer's disease models.

Molecular Therapy · Jul 1, 2023Peer-reviewedAnimal Behavior & Cognition

Elexacaftor/VX-445-mediated CFTR interactome remodeling reveals differential correction driven by mutation-specific translational dynamics

M. Kim, E. F. McDonald, C. M. P. Sabusap, B. Timalsina, D. Joshi, J. S. Hong, A. Rab, E. J. Sorscher, L. Plate

This study investigates how Elexacaftor/VX-445 affects CFTR interactome remodeling in response to specific mutations, revealing differential correction mechanisms.

bioRxiv · Feb 4, 2023Cell Biology

Distinct proteostasis states drive pharmacologic chaperone susceptibility for Cystic Fibrosis Transmembrane Conductance Regulator misfolding mutants

E. F. McDonald, C. M. P. Sabusap, M. Kim, L. Plate

The paper investigates why different CFTR mutations respond variably to the pharmacological chaperone VX-809 by analyzing proteostasis interactions and degradation pathways.

bioRxiv · Sep 9, 2021Cell Biology