Author

G. Quiroz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Animal Behavior & Cognition· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. G. Palacios· 1 shared A. Gutierrez· 1 shared A. Lovy· 1 shared A. O. Ardiles· 1 shared A. Puebla-Huerta· 1 shared A. Zakarian· 1 shared B. Morales· 1 shared C. Casanova-Canelo· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperAnimal Behavior & Cognition

A. G. Palacios

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Calcium (Ca2+) fluxes at Mitochondria-ER Contact Sites (MERCS) are a new target of senolysis in Therapy-Induced Senescence (TIS).

A. Puebla-Huerta, H. Huerta, C. Quezada-Gutierrez, P. Morgado-Caceres, C. Casanova-Canelo, S. Linsambarth, O. Diaz-Rivera, J. A. Lopez-Dominguez, S. Rodriguez-Lopez, G. Bustos, E. Silva-Pavez, A. Lovy, G. Quiroz, C. Gonzalez-Seguel, E. Salas-Huenuleo, , J. C. Cardenas

This study shows that inhibiting ER-mitochondria Ca2+ flux is a new target for senolysis in therapy-induced senescence.

bioRxiv · Mar 31, 2024Cell Biology

The unfolded protein response transcription factor XBP1s ameliorates Alzheimer`s disease by improving synaptic function and proteostasis

C. Duran Aniotz, C. Rivera Krstulovic, N. Poblete, A. O. Ardiles, M. L. Diaz Hung, C. M. P. Sabusap, Y. Gerakis, F. Cabral Miranda, J. Diaz, M. Fuentealba, E. Munoz, S. Espinosa, G. Matinez, G. Quiroz, G. Tamburini, , C. Hetz

This study shows that overexpressing the active form of XBP1 in neurons reduces amyloid deposits and improves synaptic function in Alzheimer's disease models.

Molecular Therapy · Jul 1, 2023Peer-reviewedAnimal Behavior & Cognition

Aβ oligomers trigger necroptosis-mediated neurodegeneration via microglia activation in Alzheimer's disease.

N. Salvadores, I. Moreno, N. Gamez, G. Quiroz, L. Vegas, M. Escandon, S. Jimenez, J. Vitorica, A. Gutierrez, C. Soto, F. Court

This study shows Aβ oligomers induce necroptosis in neurons via microglia activation, leading to neurodegeneration in Alzheimer's disease, and suggests necroptosis inhibition as a potential therapeutic target.

bioRxiv · Aug 27, 2021Cell Biology